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Mutations in ESR1 are the most common mechanism of acquired resistance to treatment with an aromatase inhibitor plus a cyclin dependent kinase 4 and 6 CDK4 6 inhibitor Limited evidence exists to support the simultaneous initiation of sodium glucose cotransporter 2 inhibitors and finerenone a nonsteroidal mineralocorticoid receptor antagonist in

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Mutations in ESR1 are the most common mechanism of acquired resistance to treatment with an aromatase inhibitor plus a cyclin dependent kinase 4 and 6 CDK4 6 inhibitor


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